This Article  • References  • Full text PDF  • Reply to article  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Citation map  • Contact me when this article is cited  Related Content  • Similar articles in this journal

RUSSELL N. DeJONG, M.D.

Arch Neurol Psychiatry. 1941;46(6):1068-1075.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

It has been known for several years that deficiency of vitamin E in the diet is responsible for the production of certain changes in the muscular and neuromuscular systems of experimental animals. Conditions reported to have resulted from such a deficiency are as follows: muscular dystrophy in the rat, guinea pig, rabbit and dog¹; myopathies, involving especially the nonstriated musculature, in the duck and turkey²; encephalomalacia, pareses and cerebellar disorders in the chick³; various paralyses in the suckling rat,⁴ and pareses in the adult rat, with involvement of the posterior columns, pyramidal tracts and anterior horn cells of the spinal cord.⁵ These disturbances can be prevented, and even relieved, by adding to the animal's diet the deficient vitamin or alpha tocopherol, an alcohol having the properties of vitamin E.⁶ This substances was first isolated from wheat germ oil, but can now be produced synthetically. . . . [Full Text PDF of this Article]

Author Affiliations
ANN ARBOR, MICH.

From the Department of Neurology, University of Michigan Medical School and the University Hospital.

Footnotes
Read before the Section on Nervous and Mental Diseases at the Ninety-Second Annual Session of the American Medical Association, Cleveland, June 5, 1941.