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PAUL C. BUCY, M.D.; KARL H. PRIBRAM, M.D.

Arch Neurol Psychiatry. 1943;50(4):456-461.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

In recent years, as the result of new evidence which has been presented, there has been a renewed interest in the question of the effect of the cerebral cortex on various autonomic, or vegetative, functions. Although direct evidence as to the influence of the cortex on vasomotor activity, intestinal activity, respiration, etc., has been obtained, evidence as to the influence of the cerebral cortex on sweating is meager. It is true that hundreds of cases have been observed in which a cerebral lesion resulted in hemiplegia associated with unilateral edema, vasodilatation and sweating. There has always been doubt as to whether this localized sweating was the direct result of the cerebral lesion or came about only because of the inactivity of the paralyzed extremities. Instances in which localized sweating has occurred as a result of cerebral stimulation or irritation are indeed few. The present case is an exceptionally clear example . . . [Full Text PDF of this Article]

Author Affiliations
CHICAGO

From the Chicago Memorial Hospital, and the Department of Neurology and Neurological Surgery, the University of Illinois.