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E. A. BERING, JR., M.D.; B. SALIBI, M.D.

A.M.A. Arch Neurol Psychiatry. 1959;81(6):693-698.

	Since this article does not have an abstract, we have provided the first 150 words of the full text PDF and any section headings.

The relationship of increased cerebral venous pressure to ventricular enlargement and hydrocephalus has never been satisfactorily worked out. The interdependence of intracranial pressure and cerebral venous pressure has been recognized both in clinical problems and in experimental studies, but the possibility of cerebral venous pressure as a primary cause of ventricular enlargement has remained a controversial issue.

Dandy² stated that the occlusion of the vein of Galen would cause hydrocephalus. Others have tried this and other types of experimental venous occlusions, with varying results. Russell,⁴ in her monograph on hydrocephalus, reviewed much of this work, including some experimental work of her own, and concluded that, while hydrocephalus had apparently been produced in some instances, the results were, in general, equivocal. These experimental attempts to produce hydrocephalus from increased venous pressure were all based upon local occlusion of a cerebral vein or dural sinus. This allowed the possibility that . . . [Full Text PDF of this Article]

Author Affiliations
Boston

From the Neurosurgical Research Laboratory of the Children's Medical Center.

Footnotes
Accepted for publication Sept. 23, 1958.

This investigation was supported by U. S. P. H. S. Grant B-157(C5) from the National Institutes of Health, U. S. Public Health Service.